Wnt Signaling Molecules in Left Ventricular Remodeling

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Wnt Signaling Molecules in Left Ventricular Remodeling

Under acute or chronic stresses, the adult heart undergoes a remodeling process that involves cardiomyocyte hypertrophy accompanied by apoptosis, necrosis, and fibrosis that lead to impaired cardiac contractility. The role of endogenous regeneration in this process is currently under investigation. Sustained deleterious stimuli will lead to a decompensated form of hypertrophy often culminating ...

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Editorial Commentary Wnt Signaling Molecules in Left Ventricular Remodeling Focus on Dishevelled 1

Under acute or chronic stresses, the adult heart undergoes a remodeling process that involves cardiomyocyte hypertrophy accompanied by apoptosis, necrosis, and fibrosis that lead to impaired cardiac contractility. The role of endogenous regeneration in this process is currently under investigation. Sustained deleterious stimuli will lead to a decompensated form of hypertrophy often culminating ...

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Wnt signaling molecules in left ventricular remodeling: focus on dishevelled 1.

Under acute or chronic stresses, the adult heart undergoes a remodeling process that involves cardiomyocyte hypertrophy accompanied by apoptosis, necrosis, and fibrosis that lead to impaired cardiac contractility. The role of endogenous regeneration in this process is currently under investigation. Sustained deleterious stimuli will lead to a decompensated form of hypertrophy often culminating ...

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Wnt Signaling Molecules in Left Ventricular Remodeling Focus on Dishevelled 1 Laura

Under acute or chronic stresses, the adult heart undergoes a remodeling process that involves cardiomyocyte hypertrophy accompanied by apoptosis, necrosis, and fibrosis that lead to impaired cardiac contractility. The role of endogenous regeneration in this process is currently under investigation. Sustained deleterious stimuli will lead to a decompensated form of hypertrophy often culminating ...

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ژورنال

عنوان ژورنال: Hypertension

سال: 2010

ISSN: 0194-911X,1524-4563

DOI: 10.1161/hypertensionaha.109.143297